Does Glutathione Work For Stomach Disease, Pancreatitis, and Inflammatory Bowel Disease?
Glutathione (GSH) has a very positive role in assisting with all stomach conditions, including but not limited to Gastritis, Peptic Ulcers, Stomach Cancer, Gastroenteritis, Colitis, Inflammatory Bowel Disease, Ulcerative Colitis, Crohn's Disease, Malabsorption Syndromes, and Pancreatitis.
Note what Dr. Jimmy Gutman has to say in his latest bestseller, "Glutathione - Your Key To Health" in his chapter on Digestive Diseases:
"The digestive tract is a string of connected organs stretching from the mouth to the bowels. With it we eat, digest, and eliminate waste. The many digestive disorders are caused by such factors as genetics, stress, toxins, infectious diseases and pharmaceutical drugs. This chapter discusses the latest research on glutathione in the digestive tract.
GSH helps protect the mouth and salivary organs from periodontal disease, stomatitis and gingivitis and the esophagus from inflammation. In the stomach, it protects against gastritis, peptic ulcer and cancer and in the liver, hepatitis and organ failure. GSH also defends the pancreas against inflammation and the large intestine (bowel) against colitis, inflammatory bowel disease, ulcerative colitis, Crohn's disease and cancer...
Research evidence suggests that glutathione defends the stomach lining against various threats, including toxins, oxidative stress and carcinogenesis. The results have prompted others to seek ways to raise glutathione levels in humans for both preventive and curative purposes. Elevated glutathione levels may protect against gastritis, ulcer and cancer and can certainly complement conventional treatments for these diseases.
With stomach disease, the high levels of oxidative stress and the depletion of glutathione in pancreatitis is well documented and scientists are investigating the role of antioxidant therapy in the treatment of pancreatitis and prevention of recurrent bouts. Even though antioxidant therapy is a safe complementary treatment for chronic pancreatitis, its wider adoption as a standard healthcare tool will take time.
The lynchpin of this new approach is the search for tools to enhance (modulate) intracellular glutathione levels. As these tools emerge, further research will be needed to use them effectively.
An imbalance in the formation of free radicals and a poor supply or availability of antioxidant micronutrients may cause or encourage tissue injury in inflammatory bowel disease. Levels of glutathione and its related compounds are significantly lower in these diseases.
Different antioxidants including GSH, GSH monoesters, NAC (N-acetylcysteine), Vitamin C (ascorbate), Vitamin E (tocopherol), SOD (superoxide dismutase) and others have been used with varying success.
It may not be clear whether GSH loss is a cause or consequence of these inflammatory disorders, but in either case, they are positively affected by therapies that raise or sustain GSH levels. Recent research suggests that raising glutathione levels may be a novel approach to the treatment of ulcerative colitis and Crohn's Disease."
You can also read Debbie's story with glutathione and ulcerative colitis here.
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